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don't understand, on a molecular level, how shutting down RAF/MEK will stop KRAS-driven signaling. yes, I know it's downstream of RAS but wouldn't shutting down RAF/MEK in KRAS cancers affect healthy cells?
don't understand, on a molecular level, how shutting down RAF/MEK will stop KRAS-driven signaling. yes, I know it's downstream of RAS but wouldn't shutting down RAF/MEK in KRAS cancers affect healthy cells?